Stem Cell Reports Resource Precise Correction of the Dystrophin Gene in DuchenneMuscular Dystrophy Patient Induced Pluripotent Stem Cells by TALEN and CRISPR-Cas9

نویسندگان

  • Hongmei Lisa Li
  • Naoko Fujimoto
  • Noriko Sasakawa
  • Saya Shirai
  • Tokiko Ohkame
  • Tetsushi Sakuma
  • Michihiro Tanaka
  • Naoki Amano
  • Akira Watanabe
  • Hidetoshi Sakurai
  • Takashi Yamamoto
  • Shinya Yamanaka
  • Akitsu Hotta
چکیده

Hongmei Lisa Li,1 Naoko Fujimoto,1,2 Noriko Sasakawa,1 Saya Shirai,1 Tokiko Ohkame,1 Tetsushi Sakuma,4 Michihiro Tanaka,1 Naoki Amano,1 Akira Watanabe,1 Hidetoshi Sakurai,1 Takashi Yamamoto,4 Shinya Yamanaka,1,2,5 and Akitsu Hotta1,2,3,* 1Center for iPS Cell Research and Application, Kyoto University, Kyoto 606-8507, Japan 2iCeMS, Kyoto University, Kyoto 606-8501, Japan 3PRESTO, Japan Science and Technology Agency, Kawaguchi 332-0012, Japan 4Department of Mathematical and Life Sciences, Graduate School of Science, Hiroshima University, Higashi-Hiroshima 739-8526, Japan 5Gladstone Institute of Cardiovascular Disease, San Francisco, CA 94158, USA *Correspondence: [email protected] http://dx.doi.org/10.1016/j.stemcr.2014.10.013 This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/3.0/).

برای دانلود رایگان متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Precise Correction of the Dystrophin Gene in Duchenne Muscular Dystrophy Patient Induced Pluripotent Stem Cells by TALEN and CRISPR-Cas9

Duchenne muscular dystrophy (DMD) is a severe muscle-degenerative disease caused by a mutation in the dystrophin gene. Genetic correction of patient-derived induced pluripotent stem cells (iPSCs) by TALENs or CRISPR-Cas9 holds promise for DMD gene therapy; however, the safety of such nuclease treatment must be determined. Using a unique k-mer database, we systematically identified a unique targ...

متن کامل

Cellular Reprogramming, Genome Editing, and Alternative CRISPR Cas9 Technologies for Precise Gene Therapy of Duchenne Muscular Dystrophy

In the past decade, the development of two innovative technologies, namely, induced pluripotent stem cells (iPSCs) and the CRISPR Cas9 system, has enabled researchers to model diseases derived from patient cells and precisely edit DNA sequences of interest, respectively. In particular, Duchenne muscular dystrophy (DMD) has been an exemplary monogenic disease model for combining these technologi...

متن کامل

Efficient Production of Biallelic RAG1 Knockout Mouse Embryonic Stem Cell Using CRISPR/Cas9

Background: Recombination Activating Genes (RAG) mutated embryonic stem cells are (ES) cells which are unable to perform V (D) J recombination. These cells can be used for generation of immunodeficient mouse. Creating biallelic mutations by CRISPR/Cas9 genome editing has emerged as a powerful technique to generate site-specific mutations in different sequences. Ob...

متن کامل

Precise Correction of Disease Mutations in Induced Pluripotent Stem Cells Derived From Patients With Limb Girdle Muscular Dystrophy.

Limb girdle muscular dystrophies types 2B (LGMD2B) and 2D (LGMD2D) are degenerative muscle diseases caused by mutations in the dysferlin and alpha-sarcoglycan genes, respectively. Using patient-derived induced pluripotent stem cells (iPSC), we corrected the dysferlin nonsense mutation c.5713C>T; p.R1905X and the most common alpha-sarcoglycan mutation, missense c.229C>T; p.R77C, by single-strand...

متن کامل

Functional correction of dystrophin actin binding domain mutations by genome editing.

Dystrophin maintains the integrity of striated muscles by linking the actin cytoskeleton with the cell membrane. Duchenne muscular dystrophy (DMD) is caused by mutations in the dystrophin gene (DMD) that result in progressive, debilitating muscle weakness, cardiomyopathy, and a shortened lifespan. Mutations of dystrophin that disrupt the amino-terminal actin-binding domain 1 (ABD-1), encoded by...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

عنوان ژورنال:

دوره   شماره 

صفحات  -

تاریخ انتشار 2015